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Molin S, Diepgen TL, Ruzicka T, Prinz JC. Pompholyx eczema as a manifestation of HIV infection, response to antiretroviral therapy. Severe dyshidrosis in two patients with HIV infection shortly after starting highly active antiretroviral treatment. Pompholyx induced by intravenous immunoglobulin therapy. Llombart M, Garcia-Abujeta JL, Sanchez-Perez RM, Hernando de Larramendi C. immunoglobulin therapy for neurologic disease. Iannaccone S, Sferrazza B, Quattrini A, Smirne S, Ferini-Strambi L. Atopy, nickel sensitivity, occupation, and clinical patterns in different types of hand dermatitis. Magina S, Barros MA, Ferreira JA, Mesquita-Guimaraes J. Dyshidrosis: epidemiology, clinical characteristics, and therapy.

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Prevalence of skin diseases among pediatric patients in Turkey. Tamer E, Ilhan MN, Polat M, Lenk N, Alli N. Photoinduced pompholyx: a report of 5 cases. A 3-year causative study of pompholyx in 120 patients. Guillet MH, Wierzbicka E, Guillet S, Dagregorio G, Guillet G. The key role of aquaporin 3 and aquaporin 10 in the pathogenesis of pompholyx. Soler DC, Bai X, Ortega L, Pethukova T, Nedorost ST, Popkin DL, et al. Filaggrin mutations may confer susceptibility to chronic hand eczema characterized by combined allergic and irritant contact dermatitis. Molin S, Vollmer S, Weiss EH, Ruzicka T, Prinz JC. The gene for a rare autosomal dominant form of pompholyx maps to chromosome 18q22.1-18q22.3. Pompholyx of the hands after intravenous immunoglobulin therapy for clinically isolated syndrome: a paediatric case. 185(11):E530.Д«razzelli V, Grassi S, Savasta S, Ruffinazzi G, Carugno A, Barbaccia V, et al. Dyshidrotic eczema following intravenous immunoglobulin treatment. Dyshidrotic eczema associated with the use of IVIg.

Pompholyx and eczematous reactions associated with intravenous immunoglobulin therapy. Gerstenblith MR, Antony AK, Junkins-Hopkins JM, Abuav R.

LIGHT ORANGE BACKGROUND HD UPDATE

Update on psychodermatological disorders. T cell receptor (TCR) interaction with haptens: metal ions as non-classical haptens. Thierse HJ, Gamerdinger K, Junkes C, Guerreiro N, Weltzien HU. Low-cobalt diet for dyshidrotic eczema patients. Letter: regression of relapsing dyshidrotic eczema after treatment of concomitant hyperhidrosis with botulinum toxin-A.

Kontochristopoulos G, Gregoriou S, Agiasofitou E, Nikolakis G, Rigopoulos D, Katsambas A. Pompholyx with bile-coloured vesicles in a patient with jaundice: are sweat ducts involved in the development of pompholyx?. Hypothetically, topical and/or systemic inhibition of the expression of aquaporins in the epidermis could contribute with the preservation of water and glycerol, decreasing the frequency and severity of pompholyx exacerbations. Owing to osmotic gradients, among other factors, the direction of water and glycerol through aquaporins is from the skin to the environment, possibly contributing with skin dehydration, even immediately after hand washing. These channels may participate in the increase of transepidermal water loss seen in atopic dermatitis and possibly in pompholyx. Aquaporin-3 and aquaporin-10 are normally expressed in the basal layer of the epidermis, and immunohistochemical staining had demonstrated their presence in all epidermal layers in patients with pompholyx. Aquaglyceroporins can transport water and glycerol. Aquaporins are channel proteins located on cell membranes that increase their permeability, in particular aquaglyceroporins. Aquaporins have been shown to be expressed in patients with atopic dermatitis and may also be related to exacerbation and chronicity of pompholyx.

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